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Advanced glycation end products (AGEs) are formed as a result of nonenzymatic glycosylation of proteins and lipids and are present in food and in the blood circulation. In diabetes, AGEs accumulate and are involved in vascular pathology. However, the long-term effects of AGEs on vascular function are still poorly understood. We have recently shown that low-dose AGEs exposure affects the blood pressure and causes small vessel dysfunction. We now investigated the effects of chronic exposure to AGEs on small-mesenteric artery (SMA) function. Male rats were fed chow enriched with 0.05% N(ε)-carboxymethyllysine (CML) for 6 weeks or distilled water as a control. Isometric contraction of isolated mesenteric arteries was examined in organ chambers. In addition, SMA blood flow was measured in anesthetized rats by transit time ultrasonic flowmetry. CML exposure did not change blood pressure, but caused a small decrease in SMA function. Mesenteric arteries exposed to AGEs showed reduced sensitivity to the vasoconstrictor phenylephrine (pD(2)) and reduced vasoconstrictor responses to calcitonin gene-related peptide (CGRP). In addition, CML exposure caused impaired

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