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Pyridoxal is known to inhibit both alcohol oxidation and NADPH regeneration in mouse liver and to increase lipid peroxidation in alcohol-treated rats. The aim of this study was to verify whether the administration of pyridoxal alone or in combination with pyridoxamine would influence alcohol consumption, blood alcohol levels, and liver pathology in mice. The influence of pyridoxal on blood levels of triglycerides and cholesterol was also investigated. A diet containing 0.1 percent ethanol was administered in drinking water for 6 weeks to 32 male and female C57Bl/6 mice. After the alcohol treatment a group of 4-6 mice receiving vehicle only and 2 groups receiving intraperitoneal injections of pyridoxal at doses of 8 or 16 mg/kg or 2 ml of placebo solution at the same doses were studied for a second time. Alcohol consumption was measured by drinking tubes. Blood levels of triglycerides, triglycerides, cholesterol, and alanine aminotransferase were determined. Compared with saline-treated controls, mice receiving 0.1 percent ethanol showed markedly higher blood ethanol and carbon dioxide concentrations and increased liver weight. No further increase of blood alcohol levels was observed in mice receiving intraperitoneal injections of pyridoxal. Compared to the group receiving only the vehicle, the animals receiving pyridoxal showed decreased liver weights and blood triglyceride levels. Administration of pyridoxal inhibited the increase in blood cholesterol levels. Administration of pyridoxal in combination with pyridoxamine was less effective than pyridoxal alone in preventing the alcohol-induced increase in blood ethanol levels. In conclusion, the results show